Which set of analyte changes would you expect for liver cirrhosis (hepatic jaundice)?

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Liver cirrhosis causing hepatic jaundice disrupts both hepatocyte integrity and liver function, so you see signs of cell injury, cholestasis, and altered nitrogen metabolism reflected in the analytes. ALT rises because damaged hepatocytes release this enzyme into the blood. ALP goes up with cholestasis and biliary involvement that can accompany cirrhosis. Ammonia accumulates since the liver can’t efficiently convert it to urea, leading to higher NH3 levels. LDH increases as a general marker of tissue damage from liver injury. Total protein can rise in chronic liver disease due to increased immunoglobulins (hypergammaglobulinemia) even though albumin may fall, so the net protein level can be up. Urea levels can be variable, but in this pattern the overall trend shown is an elevation across these analytes, aligning with the described hepatic abnormalities. This combination best fits the expected profile for hepatic jaundice from cirrhosis among the given options.