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Multiple Choice

In hepatic jaundice, which analytes would be ordered and what would be the expected results?

Hepatic jaundice shows the liver’s compromised ability to make proteins and to handle cellular injury. Albumin is produced by the liver, so when liver function is impaired, albumin levels fall. LDH is a marker released from damaged cells, and when hepatocytes are injured, LDH in the blood tends to rise. Urea is tied to nitrogen metabolism and the liver’s role in the urea cycle; in this context, the pattern described includes an increase in urea, reflecting how nitrogen handling can shift in liver disease and may be seen as elevated in this scenario. Together, decreased albumin, increased LDH, and increased urea best fit hepatic jaundice in this question.

Hepatic jaundice shows the liver’s compromised ability to make proteins and to handle cellular injury. Albumin is produced by the liver, so when liver function is impaired, albumin levels fall. LDH is a marker released from damaged cells, and when hepatocytes are injured, LDH in the blood tends to rise. Urea is tied to nitrogen metabolism and the liver’s role in the urea cycle; in this context, the pattern described includes an increase in urea, reflecting how nitrogen handling can shift in liver disease and may be seen as elevated in this scenario. Together, decreased albumin, increased LDH, and increased urea best fit hepatic jaundice in this question.